Pathophysiology of Heart Failure - primeira edição

EAN (ISBN-13): 9780792335719
ISBN (ISBN-10): 0792335716
Livro de capa dura
Livro de bolso
Ano de publicação: 1995
Editor/Editora: SPRINGER VERLAG GMBH
624 Páginas
Peso: 1,043 kg
Língua: eng/Englisch

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Página de detalhes modificada pela última vez em 2024-01-26T23:08:06+00:00 (Lisbon)
Número ISBN/EAN: 9780792335719

Número ISBN - Ortografia alternativa:
0-7923-3571-6, 978-0-7923-3571-9
Ortografia alternativa e termos de pesquisa relacionados:
Autor do livro: takeda, rob beamish
Título do livro: heart, pathophysiology

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Dados da editora

Autor: Naranjan S. Dhalla; Pawan K. Singal; Nobuakira Takeda; Robert E. Beamish
Título: Developments in Cardiovascular Medicine; Pathophysiology of Heart Failure
Editora: Springer; Springer US
578 Páginas
Ano de publicação: 1995-12-31
New York; NY; US
Língua: Inglês
299,59 € (DE)
307,99 € (AT)
330,50 CHF (CH)
Available
XLII, 578 p.

BB; Hardcover, Softcover / Medizin/Klinische Fächer; Kardiologie, Angiologie; Verstehen; cardiovascular; cardiovascular system; circulation; heart; hypertension; vascular disease; Cardiology; BC

I. Cardiomyopathy and Contractile Dysfunction.- 1. Experimental models of heart failure and cardiomyopathy.- 2. Role of tissue and circulating substance P in cardiovascular injury associated with Mg-deficiency.- 3. Functional and metabolic effects of norepinephrine on the rat heart.- 4. Interventions in experimentally induced diabetic cardiomyopathy.- 5. Diabetes as a risk factor for cardiovascular disease: Experimental models.- 6. Ethanol-induced hypertension: the role of Acetaldehyde.- 7. Abnormalities in cardiac contractile proteins and cardiac dysfunction.- 8. Role of the slow sodium channel in hereditary cardiomyopathy.- 9. Cardiac contractile failure caused by disturbances in myofibrillar energy supply and pathogenesis of cardiomyopathies.- 10. Changes in contractile proteins under oxidative stress.- 11. Antioxidants and the heart.- 12. Gene expression of cardiac myocytes: a pharmacologic target for the failing heart?.- II. Calcium-Related Events and Heart Dysfunction.- 13. Regulation of slow Ca2+ channels of myocardial cells by cyclic nucleotides and phosphorylation.- 14. Multiple roles for the membrane-associated Ca2+/calmodulin-dependent protein kinase in the regulation of sarcoplasmic reticulum function in heart muscle.- 15. Calreticulin, a multifunctional Ca2+ binding protein of smooth muscle sarcoplasmic reticulum membranes.- 16. Intracellular Ca2+ transients in response to step length changes in aequorin-injected ferret papillary muscles.- 17. Initial changes in ?-adrenergic receptor function during development of rapid ventricular pacing-induced heart failure.- 18. Impaired cellular signaling of the adenylyl cyclase and the phosphoinositide pathway in septic cardiomyopathy.- 19. Defective G-protein expression and adenylyl cyclase in genetic andexperimental hypertension.- 20. Electrophysiological studies of wild-type and mutant Na+-Ca2+ exchange proteins.- 21. Cardiac sarcolemmal Na+/H+ exchange after a myocardial infarction in the rat.- 22. Myocardial Na, K-ATPase concentration and heart failure.- 23. The cytosolic calcium-force relation of vascular smooth muscle during the contraction and the relaxation.- III. Ischemia and Cellular Interactions.- 24. Ventricular remodeling following coronary artery constriction and hypertension.- 25. Collagenous proteins in scar tissue subsequent to myocardial infarction.- 26. Structural remodeling and functional changes in chronic hypoxia-induced right ventricular hypertrophy.- 27. Mechanism of openings and role of the ATP-sensitive K+ channels during myocardial ischemia/reperfusion.- 28. The contribution of K+ currents to electrical heterogeneity across the canine ventricular wall under normal and ischemic conditions.- 29. Diabetic state reduces ischemic K+ loss and H+ efflux in isolated rat hearts.- 30. Divergent effects of receptor- and nonreceptor-mediated activators of sodium-hydrogen exchange on reperfusion-induced contractile dysfunction.- 31. Heart cells in culture for studying anoxia and “simulated ischemia” at the cellular level.- 32. Cardioprotection and basic fibroblast growth factor.- 33. The heart and nitric oxide.- 34. The endothelium and the regulation of coronary blood flow.- 35. Signal transduction in vascular smooth muscle: synergistic interactions between receptors and transduction pathways.- 36. Modulation of peak aerobic capacity by the peripheral circulation during the course of the syndrome of congestive heart failure.